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The identification of these dichotomous astrocytic states, particularly the reparative A2 phenotype, has revealed promising therapeutic targets that will be examined in later sections of this review

Critical steps include: Establishing clinical trial material manufacturing processes that guarantee consistency and scalability

Animals were constantly monitored during the period of the study

Together these molecules constitute the **reference pairing** in tendon, ligament, muscle and vascular repair research: two complementary mechanisms of action, one shared angiogenic vector, and dramatically expanded tissue coverage

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